James E Clark

Research interests involve the application of both in vivo and in vitro physiological and molecular techniques to investigate the response of the myocardium to the stresses of ischaemia, reperfusion and trophy. 1. The signaling pathways involved in carbon monoxide (CO)-mediated cardiac protection, 2. The role of the p38-MAP kinase isoforms in cardiac physiology, 3. Developing and characterizing new methodologies for measuring cardiac function in murine models.