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Synaptic Mitochondrial Dysfunction and Septin Accumulation Are Linked to Complement-Mediated Synapse Loss in an Alzheimer's Disease Animal Model.

Györffy Balázs A.,Tóth Vilmos,Semmelweis University,Gulyássy Péter,Kovács Réka Á.,Vadászi Henrietta,Micsonai András,Tóth Melinda E.,Sántha Miklós,Homolya László,Drahos László, CRU Hungary Ltd.,Kékesi Katalin A.,Kardos József

Cellular and Molecular Life Sciences（2020）

Cited 46|Views37
Key words
Synaptic pruning,Alzheimer's disease,Complement C1q,Fluorescence-activated synaptosome sorting,Mitochondrial dysfunction,Septins
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