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    • Table S4 from Hypoxic Memory Mediates Prolonged Tumor-Intrinsic Type I Interferon Suppression to Promote Breast Cancer Progression

    crossref（2024）

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    Abstract
    GSEA analysis with genes upregulated in 68H compared to 68N
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    【要点】：本文揭示了低氧记忆通过介导肿瘤内在类型I干扰素长期抑制，促进乳腺癌进展的机制，并通过GSEA分析确认了68H与68N基因表达的差异。
    

    【方法】：研究采用基因集富集分析（GSEA）来识别在低氧条件下68H细胞中上调的基因。
    

    【实验】：实验通过对比68H和68N细胞系，在低氧条件下进行GSEA分析，发现68H细胞中特定基因的上调与乳腺癌进展相关。数据集名称未在摘要中明确提及，但实验结果支持了低氧记忆与乳腺癌进展之间的联系。